The main clinical presentation is lameness linked to arthritic change in the joints. The colostrum from infected dams will contain both virusĪnd antibody, the latter affording no maternal protection – thus unlike other infections –Ĭolostrum can actually be the cause of a new infection in the kid, rather than itsĬAE infection results in a range of clinical signs, some of which have already beenĭiscussed. Response, but the antibody produced is not protective – and an infected goat is essentiallyīoth virus and antibody positive – and it is the antibody test that forms the basis of any The incubation period between infection and theĭevelopment of clinical signs can be very variable. Such is associated with a high prevalence of latent inapparent infection, in which goats The development of any clinical disease after infection will be slow and protracted, and as Hence potentially many kids – and this is also true for Johne’s disease. Herds can also lead to increased infection rates as one infected doe can infect the pool, and The practice of feeding pooled colostrum and milk in dairy Increased infection rates will occur in housed and intensively managed Minimal risk of transferring infection, both practices need to be risk managed if eradication isīeing attempted. Levels, and although both embryo transfer and artificial insemination may pose only a Transplacental infection can occur but is thought to be at low Milking parlour, or through shared use of equipment such as drenching guns To nose contact and aerosol transfer, via “infected milk impacts” at the teat end in the There are however many other important routes of infection such as via nose This knowledge is vitalĬAE is transmitted predominantly via the ingestion of infected colostrum or milk from

How the infectious agent is picked up and spread within a population. Occur, and is an important factor in CAE control programmes, that have been developed onĪ compulsive / voluntary basis in many countries around the world.Īn important part of any disease control or preventative programme, is an understanding of Although the two viruses causeĭifferent clinical presentations in their respective host species, cross species infection can Referred to collectively as SLRV (small ruminant lentivirus). Insidious conditions such as CAE and Maedi Visna (MV) of sheep, the two diseases often Both kids were infected at 5-6 and 9 months of age.“The Veterinary Guide to Goat Health and Welfare”Ī lentivirus in the family Retroviridae, a genus causing a group of slowly developing A third group of two kids was not removed from their infected dams. All four kids became infected with CAE virus they developed serum antibody to CAE virus between 5-6 and 9 months of age. A second group of four kids was removed at birth and fed heat-treated goat colostrum, followed by milk from CAE virus-infected does. One goat was positive at three and nine months of age but was negative when tested at 12 months of age. Nine of the ten goats were negative for serum antibody to CAE virus when tested at 5-6, 9 and 12 months of age. Passively acquired serum antibody to CAE virus was detected in some kids at two to three months of age. They were raised apart from infected goats with separation maintained by a wire fence. Subsequently the kids were fed reconstituted spray dried cows' milk powder. Kids were fed on goat colostrum, which had been heated to 57 degrees C for ten minutes and then held in a thermos flask for one hour. One group of ten goat kids was removed from infected does at birth before suckling or licking by the doe could occur (snatch birth technique).

Goat kids from a herd endemically infected with caprine arthritis encephalitis (CAE) virus were raised according to 3 methods.

Cae in goats testing